Category: archive
Welcome New Lab Members
Welcome to new lab members Medha, Amin and Rebecca. Delighted to have you join us and work on projects in synaptic plasticity, miniature neurotransmission and neurotransmitter logic.
Congratulations Soumya
Congratulations to Soumya on his new position as senior scientist at Epiterna. We are very happy you are staying in Lausanne and continuing to work on ageing.
Well done Wei
Congratulations to Wei on her new position at our neighboring institute of the University of Lausanne – UNIL. We are very happy you are staying in Lausanne and we look forward to collaborating.
Congratulations Jamshid
Congratulations to Jamshid on his new position as Data Manager at Clario. They could not have made a better choice.
New Review by Emma
Graduate student Emma Källstig has published a new review on the links between the NF-κB pathway and the motor neuron disease ALS. Read it here.
Welcome Greta
We are delighted to welcome Greta Limoni to our group as a postdoctoral scientist. Greta received her PhD from the University of Geneva where she worked on understanding interneuron development. Greta now turns her attention to investigating molecular and cellular mechanisms that may contribute to the motor system diseases SMA and ALS.
Interview with Jamshid
Nice interview with Jamshid about his efforts helping to form and act as the first president of the new EPFL Postdoc association.
Predictive Power
A new publication with our clinical collaborators at Columbia University Medical Center in New York reveals sensory-motor circuit dysfunction in human Spinal Muscular Atrophy (SMA) patients as was predicted by our Drosophila model of SMA. Unfortunately 4-AP treatment, which we found was efficacious in models to correct sensory-motor deficits, at lower levels does not seem (…)
Welcome Marine
We are delighted to welcome Marine Van Campenhoudt to our group as a Ph.D. student in the EDNE neuroscience program. Marine previously carried out her masters research in the lab and is working on molecular and cellular mechanisms that may contribute to motor neuron disease (ALS).